復溶 |
1.Induction of LLT1 cell surface expression by pathogens and IFN-{gamma} contributes to modulate immune responses.Germain C, Meier A, Jensen T, Knapnougel P, Poupon G, Lazzari A, Neisig A, Hakansson K, Dong T, Wagtmann N, Galsgaard ED, Spee P, Braud VM.J Biol Chem. 2011 Sep 19.2.Characterization of Alternatively Spliced Transcript Variants of CLEC2D Gene.Germain C, Bihl F, Zahn S, Poupon G, Dumaurier MJ, Rampanarivo HH, Padkjar SB, Spee P, Braud VM.J Biol Chem. 2010 Nov 12;285(46):36207-15. Epub 2010 Sep 14.3.Functional Consequences of Interactions between Human NKR-P1A and Its Ligand LLT1 Expressed on Activated Dendritic Cells and B Cells.Rosen DB, Cao W, Avery DT, Tangye SG, Liu YJ, Houchins JP, Lanier LL.J Immunol. 2008 May 15;180(10):6508-17.4.Malignant Glioma Cells Counteract Antitumor Immune Responses through Expression of Lectin-Like Transcript-1.Roth P, Mittelbronn M, Wick W, Meyermann R, Tatagiba M, Weller M.Cancer Res. 2007 Apr 15;67(8):3540-4.
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儲存溶液 |
C-type lectin domain family 2 member D, Lectin-like NK cell receptor, Lectin-like transcript 1, LLT-1, Osteoclast inhibitory lectin, CLEC2D, CLAX, LLT1, OCIL
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